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Whats more, the NOD-like receptor family pyrin domain-containing 3 (NLRP3), also upregulated by the activation of NF-B, 38 forms inflammasome and activates protein kinase C epsilon (PKC), 39 thus imparting IRS-1 40 (Fig

Proposed mechanisms include improvements in endothelial function, reduction in systemic inflammation, favourable effects on blood pressure and lipid profiles, and direct actions on cardiac and vascular tissues expressing GLP-1 receptors

This creates a vicious cycle where metabolic dysfunction perpetuates further insulin resistance

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