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Notwithstanding the compensatory mechanisms, significant changes in expression of both nuclear and mitochondrial genes were described in the mutants, suggesting that, together with its antioxidant function, PrxIIF is an important candidate for perception of changes in the redox-state in the mitochondria (Finkemeier et al., 2005)
The first responding cells are microglia and adult oligodendrocyte precursor cells (OPC), which migrate towards the lesion, with astrocyte responses to follow
Exposure to zinc oxide nanoparticles (ZnO-NPs) induces cardiovascular toxicity and exacerbates pathogenesis role of oxidative stress and MAPK signaling
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