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Pong et al., 2001), other investigators suggest that antioxidant depletion, rather than ROS production, induces cell death (Ghibelli et al., 1995
Direct activation of HSF1 by macromolecular crowding and misfolded proteins
IGF-1 LR3 offers wider, longer-lasting anabolic benefits than shorter-acting IGF-1 versions, which can necessitate frequent dosage or target localized applications
It can attack polyunsaturated fatty acids (PUFAs) and produce lipid peroxides (lipid-OOHs) and their reactive degradation products [72, 75]
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