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By bolstering synaptic resilience while reducing the accumulation of misfolded proteins, these therapies touch on another hallmark of brain aging: the loss of proteostasis
Nevertheless, as mentioned above, p62 also regulates cell survival via the NRF2 antioxidant response pathway: in particular, the interaction between p62 and KEAP1 is essential for p62-dependent NRF2 signalling [199, 248, 249]
Wilding John PH, Batterham Rachel L, Calanna, Salvatore, et al
Molecular Metabolism , 46 , 101131
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