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Beyond glycaemic control, GLP-1 medications slow gastric emptying , which prolongs the sensation of fullness after meals and contributes to reduced appetite
Consequent mechanistic studies revealed that L-theanine, a glutamate analogue, competes with glutamate to bind the glutamate receptor, resulting in suppression of chemotherapy efflux (extracellular transport), increasing their concentration (27)
It activates PI3K/Akt and MAPK pathways, preventing neuronal death caused by amyloidogenesis, glucose deprivation, neuroinflammation, and apoptosis (8)
Brain insulin resistance may indeed play a role in the pathophysiology of cognitive disorders, and addressing this may be a mechanism through which GLP-1RAs act pro-cognitively 35
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