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For instance, the increase in OS through an augment in defective mitochondria and an inefficient EAS trigger and maintain a specific and chronic inflammatory response in vitiligo patients (via IFN-y CXCL10 CD8 + T cells) (Xie et al., 2016)

10.1093/hmg/ddv451 37 WrightJ

V., & Goosens, K

Here, we defined a novel NRF2 target gene set linked to NRF2-dependency in cancer cell lines, and observed that a significant portion of these genes is devoid of promoter-proximal NRF2 occupancy

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