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Because the vast array of molecular and cellular mechanisms modulated by these two new drug classes often intercept in pathophysiological background of chronic cardiovascular and neurodegenerative diseases, it is possible that any future new anti-diabetic drug may be also relevant for cardiometabolic and cognitive decline, independently from the anti-diabetic action
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Ayaori, M., et al
MIF null mice are entirely resistant to the detrimental effects of OVX, implying that MIF acts as a key downstream mediator of both the detrimental effects of estrogen deficiency and the beneficial effects of estrogen replacement on skin wound healing [57, 175, 182]
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