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DHHC11 and APT1 coregulate the palmitoylation status of its C-terminal residues (Cys1704, Cys1713, and Cys1714), which controls its anchoring to and release from the ER and thereby mediates lipid delivery to the expanding phagophore [138]

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Regular NAC supplements are cheaper and more stable, but depend on a well-functioning conversion process

It participates in the Keap1/Nrf2/ARE biological pathway the primary mechanism by which sulforaphane exerts its beneficial effects