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Nitric-oxide (NO), VEGF / VEGFR2 and FAK-paxillin pathway activity in soft-tissue-repair models
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Mechanistically, activation of GLP-1R by GLP-1 or exenatide phosphorylates STAT3, which drives human monocyte-derived macrophages to develop an M2 phenotype marked by increased expression of CD163, CD204, and IL-10 [86]
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