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These are separate processes mediated by GLP-1 receptors in different organs
To determine if this mechanism of action is responsible for the mgtCBR regulation phenotype, we exposed Salmonella to 5OP but countered the acidity by adding NaOH
Assuming a preferential central action of GLP1/E, we conducted label-free, quantitative proteomic analyses in the hypothalamus of the two models of PCOS, after chronic treatment with the di-agonist, as a means to disclose putative pathways for the metabolic actions of the di-agonist, and the basis for the partially differential responses between the two models of PCOS, which diverge also in terms of phenotypic presentation
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