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In control animals, surgical detachment resulted in definitive healing failure with impaired walking and permanent knee flexure persisting through 90-day follow-up
BPC-157 primarily engages angiogenic signaling (VEGF pathway modulation) and nitric oxide system regulation, addressing vascularization limits in tendon healing
New blood vessel formation brings oxygen, nutrients, and immune cells to injured tissues, addressing the fundamental bottleneck in tendon and ligament healing where poor vascularization slows repair
A stronger immune system is vital after surgery to protect against infections and speed up recovery
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