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doi: 10.1046/J.1365-2133.2003.05417.X 156 Bengtsson, LundbergMAvila-CarioJJacobssonGHolmgrenAScheyniusA
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Results clearly present the chemical potentiation of LASSS on physiological functions of HGF, as revealed by enhanced receptor-binding affinity and nitration-dysfunction resistance through interaction/reaction with LASSS under physiological conditions
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