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For hair specifically, the Pyo 2007 study (PMID 17703734) described a more defined mechanism: in cultured human dermal papilla cells, AHK-Cu increased proliferation, raised vascular endothelial growth factor (VEGF), lowered transforming growth factor-beta1 (TGF-beta1), and shifted apoptosis markers toward cell survival
supports DNA repair & mitochondrial biogenesis [1][2][3] MOTS-c 10 mg Mitochondria-encoded peptide
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