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ROS not only contributes to chronic liver inflammation but also potentiates carcinogenic processes by promoting lipid peroxidation and genomic instability (Uchida et al., 2020)
Long-term inflammation and H 2 O 2 /O/OH/ 1 O accumulation may damage the DNA of ESCs, increasing the risk of skin cancer [299]
Molecular hydrogen interacted with the cross talk of the HO-1/IL-10 axis and suppressed the expression of phosphorylated STAT3 and the activation of downstream pMAPK signaling to reverse hepatocyte apoptosis as well as hepatic inflammation and fibrosis in NASH specimens [30]
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