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When heat stress exceeds a critical threshold, excessive ROS burden and mitochondrial damage may indirectly lead to the structural and functional inactivation of HSPs (Zhao et al., 2021)
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Studies on knockout and overexpression strains of flv2/flv4 suggest the existence of an alternative electron transport pathway to an unidentified acceptor near the Q B site (Santana-Sanchez et al., 2019)
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