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It was a population of older postmenopausal women, a meaningfully different biological context, in whom initiating hormone therapy may carry different risks than initiating it earlier [11]
[12] All patients should be evaluated by a healthcare professional to ensure none of the above contraindications or precautions apply
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an environmental pollutant)-induced lipid peroxidation and cell necrosis by blocking CYP2E1-mediated CCL 4 metabolic activation and by upregulating Nrf2 downstream genes for NQO-1, HO-1, GCL, GST, and superoxide dismutase (SOD1) (67, 68)
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