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CREB activation induces adipogenesis in 3T3-L1 cells

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Baseline scores for each dimension are shown for the two treatment groups with respect to the population norm of patients with chronic disease from the Medical Outcomes Study (31) in Figure 5

[4] The scientists note that over the course of the study, VIP would emerge as an endogenous modulator of pulmonary vascular remodeling and inflammation, through its suppression of NFAT activation. In particular, NFAT suppression may play an important role in preventing pulmonary fibrosis, the end stage of diverse inflammatory conditions such as COPD, sarcoidosis, etc