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Although it is well established that the endogenous GLP-1R system is essential for glycemic control, the specific GLP-1R population that mediates this effect is still a matter of debate
GHRP-2, as a full agonist, drives the receptor above this baseline, but the existence of constitutive activity is part of why the ghrelin/GHS-R1a axis behaves as a rheostat on GH output rather than a simple on/off switch
found that GLP-1 and exendin-4 could completely protect cultured rat hippocampal neurons against glutamate-induced apoptosis (191)
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