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The widespread distribution implicates GLP-1 in several neural processes, including neuroprotection, synaptic plasticity, and modulation of neuroinflammation [7]
This circadian rhythm is regulated by food intake, and specifically, secretion and biological action of gut derived peptide hormones, including glucose-dependent insulinotropic polypeptide (GIP), glucagon-like peptide-1 (GLP-1), and glucagon-like peptide-2 (GLP-2)
They demonstrated that the B-chain C-terminal (BC-CT) residues (GlyB20-ThrB30) act as a zipper with a hinge at the PheB24 residue
Our preliminary data suggest that GLP-1s suppress intake through a different mechanism. The drugs slow gastric emptying, which can lead to a slower rise in blood alcohol
