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Curr Neuropharmacol 19(8):1202
Multiple Sclerosis Considering the potential of GLP-1RAs for the treatment of NDD, multiple sclerosis, a chronic progressive autoimmune disease caused by autoantibodies that stimulate inflammation and destruction of the myelin sheath of neuronal axons, disrupting axonal conduction and function should also be mentioned
thus, the resultant KEAP1 mutant proteins are not able to fulfill the adaptor function of the E3 ubiquitin ligase for NRF2
We report a DKA rate at 5 years of 2.1%
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