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The results showed that the six MORF2s-GFP proteins accumulated to much higher levels in the presence of SsCm1 than that of the co-expression with SsVSP25, while GmPETE, another SsCm1-interacting protein, accumulated at the similar level when co-expressed with SsCm1 or SsVSP25 (Fig

Cells then recycle it back using an enzyme (glutathione reductase), creating a powerful protection cycle

Thus, the neuroprotective effects exhibited by curcumin in preclinical TBI models can be attributed to its ability to modulate oxidative stress, neuroinflammation, mitochondrial dysfunction, and autophagy

In pernicious anaemia, the immune system mistakenly attacks the gastric parietal cells that produce intrinsic factor, leading to B12 deficiency over time