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It is essential in the production of cellular energy due to its role as a co-factor in the Krebs cycle
Its simultaneous activation of three receptor pathways GLP-1R for appetite suppression and insulin secretion, GIPR for augmented insulin response and central appetite modulation, and GCGR for energy expenditure and hepatic fat mobilisation produces metabolic effects that exceed what single or dual agonism can achieve [4][8][9]
BPC 157 also increased cell survival after H2O2 stress 1,2
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