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In murine studies, deletion of the IGF-1R in skeletal muscle resulted in glucose intolerance issues ultimately leading to T2D at an early age, because, although they express insulin receptors, they cannot form hybrid or IGF-1 receptors [162]

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Collectively, current findings indicate that PDEVs represent biocompatible, multi-target agents that effectively attenuate inflammation and promote tissue repair by overcoming the barriers to drug delivery and toxicity limitations of conventional therapies