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See World Health Organization (2018, Table A5.5)

Chicago Smith, Yolanda

63 Prolonged skeletal muscle inactivity promotes mitochondrial dysfunction, resulting in increase in mitochondrial ROS production and leading to protracted oxidative stress and disturbed redox signaling, 64 which can exacerbate IR through multiple pathways such as JNK and NF-B pathways (Fig

For example, to signal emesis after ingestion of a potentially harmful substance, 5-HT is released from ECs into the bloodstream, traveling to the area postrema of the nucleus tractus solitarius to induce vomiting ( Table 1 Enteroendocrine nomenclature

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