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(73) reported that in hepatocytes, L-carnitine inhibited stress-induced transcription factor early growth response protein 1 (EGR1), subsequently downregulating its target gene NEDD9 and modulating the NEDD9/FAK/AKT pathway, signaling pathway, indicating a potential protective mechanism in that experimental context
doi: 10.3791/52434-v 81 SheerinA
It is preferable to use combination therapies having complementary mechanisms of action that target different pathways addressing the multiple pathophysiologic abnormalities of T2DM [10,11,12]
Macrophage polarization induced by probiotic bacteria: a concise review
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