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In addition, an increasing amount of research has supported a pathophysiological role for ferroptosis in the development of cardiovascular diseases, including myocardial ischemiareperfusion injury, myocardial infarction, adriamycin-mediated myocardial injury, and heart failure [69]
(14) However, direct experimental data evaluating the combined implication of Mod GRF (1-29) and Ipamorelin is currently lacking
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