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[10] Possible drug target [edit] Triple-negative breast cancer cells rely on glutathione-S-transferase Pi1, and inhibitors are being studied
Cells were centrifuged and resuspended in 0.1 M sodium cacodylate buffer and post-fixed for 1 h with 1% osmium + 0.1 M sodium cacodylate buffer, then dehydrated and embedded in Agar 100 resin
Experimental findings indicate enhanced resistance of the mucosal lining to chemical or drug-induced injury, suggesting involvement in barrier maintenance and localized repair mechanisms
In some cases, levels exceed 3,000 pg/mL
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