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These effects are attributable to redox regulation by SFN, since (i) SFN induced ROS and in parallel GSH depletion in primary human T-cells and (ii) thiol-containing antioxidants abolished the immunosuppressive effect of SFN
GonalvesAAGarciaARRolim FilhoSTda SilvaJARde MeloDNGuimaresTCet al
Higher-likelihood alternative causes: In UK clinical guidance, GSM is a common and under-treated driver of dryness, dyspareunia and tissue fragility, and should be actively assessed, particularly in peri-/post-menopausal patients or those with iatrogenic hypo-oestrogenism
The risk is generally highest during the initial weeks of treatment and following dose increases, with symptoms often improving as the body adjusts to the medication
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