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Targeting mitochondrial iron metabolism suppresses tumor growth and metastasis by inducing mitochondrial dysfunction and mitophagy
It transports long-chain fatty acids into the mitochondria, the powerhouse of cells, where these fats are oxidized and used to create energy
This increased usage has created challenges for some people with T2D who have struggled to access certain GLP-1 drugs, as reported by NPR in 2024
Tohgi H, Abe T, Yamazaki K, Murata T, Ishizaki E, Isobe C
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