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Protective effects of diallyl disulfide on carbon tetrachloride-induced hepatotoxicity through activation of Nrf2
In addition to the relevance of POP conjugation cited previously, hyperglycemia and hyperinsulinemia appear to directly inhibit glutathione synthesis (GCL), and the increased production of reactive oxygen species which accompanies diabetes further depletes GSH levels
10.1136/bmj.n2103 23 CooneyL
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