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Conventional oral B12 formulations often face significant potential challenges, including its primary reliance on IF for absorption in the Ileum, and potential degradation by stomach harsh acidic envionment, and gastric enzymes
Epigenetic and Cellular Reprogramming: By blocking NNMT-mediated methylation of nicotinamide, preserves NAD salvage pathway activity, increases SIRT-1 signaling, reduces histone deacetylation, and promotes metabolic reprogramming toward oxidative phosphorylation and glycolysis balance, supporting tissue repair and energy homeostasis 1-4
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Clinical studies have confirmed that AOD-9604 does not stimulate IGF-1 production in any biological system tested, including human clinical trials monitoring serum IGF-1 levels over 24-week treatment periods
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