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, 2020, vol
In cerebral ischemia-reperfusion models, riboflavin attenuates NOX2-dependent superoxide generation, lessens cytochrome c release, and inhibits apoptosome activation
These substances, such as IL-6, IL-8, C-X-C motif chemokine ligand (CXCL) neutrophil chemokines, and TNF-, act on endothelial cells adjacent to blood vessels, attracting and recruiting neutrophils to the site of injury (106, 107)
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