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Reduced oxygen and glucose delivery lowers adenosine triphosphate (ATP) production, impairs Na+/K+-ATPase activity, depolarizes cell membranes, and promotes excitatory neurotransmitter release that can exacerbate neuronal injury [2,3,4]
The results support the theory that the balance between inhibitory and excitatory signals is altered in autism
Here is what the data showed
Severe hyperhomocysteinemia in patients is able to develop neurological and cardiovascular disorders as well as premature death owing to complications [106]
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