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The clinical value of most of the so far described Nrf2 activators however was rather limited, mostly due to undesired interactions of these electrophiles with other cellular targets
Despite the exciting therapeutic potential of these multi-receptor agonists, the causal effects of activating the GLP-1R, GIPR, and GCGR pathwaysboth individually and in combinationon MASLD and its multi-organ complications are not yet clearly established [21, 23, 24]
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