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Its primary mechanism of action involves increasing the expression of Brain-Derived Neurotrophic Factor (BDNF) in the cerebral cortex and hippocampus, promoting neuroplasticity, the formation of new synapses, and neuronal survival

Diallyl disulfide and diallyl trisulfide suppress oxidized LDL-induced vascular cell adhesion molecule and E-selectin expression through protein kinase A- and B-dependent signaling pathways

Regionally, North America and Europe lead in consumption, accounting for over 60% of the global market

Researchers use GHK-Cu as a copper-binding peptide model for studying peptide signaling, gene expression, and extracellular-matrix-related pathways