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CrossRef Editorial on Genome-wide interaction study with body mass index identifies CYP7A1 and GIPR as genetic modulators of metabolic dysfunction-associated steatotic liver disease Julia Kozlitina Clinical and Molecular Hepatology .2026
Moreover, in some cases, the numbers in the subgroups were small
In addition, it has long been known that GSH is capable of forming disulfide bonds with cysteine residues of proteins, and the relevance of this mechanism (S-glutathionylation) in regulation of protein function has been well documented in a number of research fields
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