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The additional 2-2.5 kg of loss comes predominantly from fat tissue, reflecting the lipolytic and anti-lipogenic mechanism
P21 reversal of TBI-induced dendritic and synaptic density loss occurred over treatment periods measured in weeks to months
Amylin and GLP-1 both curb appetite, but through partly separate brain circuits amylin acts strongly via the area postrema and hindbrain, GLP-1 through its own receptor pathways
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