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Combined with the fact that semaglutide already has approval for use in Type 2 diabetes mellitus and a good safety record, there is potential for semaglutide and other GLP-1R agonists to be repurposed as neuroprotective therapeutic agents

GLP-1(9-36) binds at an atypical site formed by the upper halves of TM1 and TM2, extracellular loop 1, and the extracellular domain structurally distinct from the canonical peptide-binding site and induces partial receptor activation that is pharmacologically potentiated by the orally active allosteric modulator LSN3318839 (Li et al., 2024)

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