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The possible mechanisms are as follows
New-User design and landmark analysis A new-user design restricted the population to patients without prior SGLT-2 inhibitor or GLP-1 RA exposure before the index date [30]
Enhancing insulin secretion in response to food GLP-1s stimulate the pancreas to release insulin only when blood sugar levels are elevated, reducing the risk of hypoglycemia (low blood sugar)
Despite localization of mGIPR-Ab/P3 predominantly in CVOs, and not in deeper parenchyma of the brain, c-Fos activation was observed in multiple neural substrates involved in food intake control 21,22,23,24,25,26,27,28,29,30 (Fig
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