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identified proteins that are critically relevant in T21 pathogenesis and are involved in iron homeostasis (ceruloplasmin and transferrin), lipid metabolism (zinc-a2-glycoprotein, retinol-binding protein 4, and apolipoprotein A1), and inflammation (complement C9,a-1B-glycoprotein, collagen alpha-1 V chain) (Perluigi et al
Coester B, Koester-Hegmann C, Lutz TA, Le Foll C
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