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Their clinical trials (NCT04542161) conclusively demonstrated that ME/CFS patients suffer from a significant, localized deficit of glutathione in the cerebral cortex, directly correlating with the severity of their neuroinflammation and cognitive dysfunction

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A decline in ACE2 function in aged individuals, coupled with the age-associated deterioration in mitochondrial functions results in chronic metabolic disorders like diabetes or cancer, and predisposes the host for increased susceptibility to infection, vulnerability to health complications, and intensifies the risk of mortality 163

How Can Patients Minimize Risks During Treatment