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In laboratory animals, the forced overexpression of the TKT gene promotes the development of MAFLD as well as the advancement of MAFLD to the more severe state referred to as metabolic dysfunction-associated steatohepatitis (MASH)
A similar finding is plausible for retatrutide given similar albumin-binding kinetics, but prescribers should await phase 3 subgroup data before assuming equivalent behavior in decompensated cirrhosis
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A rapid increase in the total number of cell surface functional GABAA receptors induced by brain-derived neurotrophic factor in rat visual cortex
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