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[7] In isolated perfused colon, butyrate and propionate dose-dependently stimulated GLP-1 secretion, [8] and studies in human enteroendocrine cells confirmed that SCFAs drive gut-hormone production in human (not just rodent) tissue

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Physical inactivity, by contrast, promotes mitochondrial stagnation, impaired fatty acid oxidation, and chronic oxidative stress, further depleting glutathione reserves

A RECENT population-based analysis has highlighted potential behavioural side effects associated with Glucagon-like peptide 1 receptor agonists (GLP1RA)