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The mechanism likely involves correction of oxidative stress and modulation of glutamate signaling via the cystine-glutamate antiporter (system Xc-) [73][74]
Inhibiting the elevated expression of GJ components in hepsin / mice reduces APAP toxicity by limiting oxidative stress in the liver To investigate GJ expression variations after APAP administration in wild-type and hepsin / mice, immunofluorescence staining for Cx32 was assessed at different time points
Cch GABA gip bn gim cm gic mt mi Li ch ca GABA Cch tng nng GABA mt cch t nhin GABA L G
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