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Perkins GD et al (2018): A Randomized Trial of Epinephrine in Out-of-Hospital Cardiac Arrest

Constitutive activation of liver kinase B1/AMP-activated protein kinase (LKB1/AMPK) pathway in KO livers The resistance of KO mice to steatosis induced by chronic EtOH consumption suggests low hepatic GSH elicits a protective metabolic adaption in the liver

doi: 10.1016/S0140-6736(20)32511-3 Summary Keywords chronic liver disorder, metabolic syndrome, non-alcoholic fatty liver disease, oxidative stress, oral glutathione Citation Santacroce G, Gentile A, Soriano S, Novelli A, Lenti MV and Di Sabatino A (2023) Glutathione: Pharmacological aspects and implications for clinical use in non-alcoholic fatty liver disease

Among various indicators, glutathione reductase (GR), a key enzyme in the oxidative stress process, shows activity changes that can signal hepatocyte metabolic stress and functional abnormalities earlier, providing an important diagnostic window for identifying early liver damage [3]