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Notably, among the genes upregulated by MOTS-c, several are closely associated with fatty acid transport ( Cd36 ) 70 and with beta-cell communication and insulin secretion ( Efna5 and Epha5 ) 22 (Fig
8 The prevalence of vitamin C deficiency in gastric diseases is currently attributed to four mechanisms: insufficient vitamin C intake, decreased vitamin C absorption, increased metabolic requirement for ascorbic acid in gastric diseases, and increased destruction of vitamin C in the diseased stomach
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