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One important caveat: wound serum from chronic wounds (such as diabetic ulcers) contains enzymes that break down GHK-Cu, which may limit topical efficacy on infected or chronic wounds
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Experimental vascular studies indicate that BPC-157 modulates endothelial nitric oxide synthase activity by altering the interaction between endothelial nitric oxide synthase and its regulatory binding partner caveolin-1 [1][2]
Bacteria produce enzymes that break peptide bonds
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