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Previous results showed that microglial/microphage polarisation to the proinflammatory M1 phenotype in the cerebral I/R model was attributed to oxidative stress, upregulation of NF-KB and ERK (Gaire et al
BPC-157 may also upregulate the expression of early growth response gene-1 (EGR-1), which is a growth factor with potential benefits for the nervous system, having been suggested to aid in early extracellular matrix (collagen) formation [5]
Compounded medications have not been evaluated or approved by the FDA for safety, efficacy, or quality
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