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However, sustained inflammation promotes BBB disruption by inducing microglial-led phagocytosis of astrocytic end-feet [62]
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These engineered macrophages home to tumor sites, release pexidartinib to inhibit CSF-1 receptor signaling and reduce MDSC accumulation, and themselves contribute to antitumor immunity through their M1 phenotype
Antiparanodal antibodies and IgG subclasses in acute autoimmune neuropathy
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