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Description

This imbalance of antioxidants in TED patients is due to the growth of orbital fibroblasts, synthesis of autoantibodies, degradation of preadipocytes to adipocytes, and secretion of endogenous cytokines (TNF, IL1-, and IFN), which consequently leads to fibro-adipose tissue development and inflammation of the extraocular muscle

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A10413.14), which was maintained throughout the culture period

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